Cardiology 2018-Cardiac Imaging

Cardiology 2018 cardic image

Cardiac Imaging is one of the important branch in the field of Cardiology. Cardiology 2018 is a global conference scheduled in Amsterdam on September 17-18, 2018.

PULSUS invites all the research scholars and innovators from the field of Cardiology to be part of the conference and thus be updated from other research and also share your research thus helping to learn and teach more in the field of Cardiology.


Abstract submission link:


Call for Abstracts


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Send us your abstracts and be a part of the global congress where eminent personalities from the field of Cardiology meet up.

September 17-18, 2018in Amsterdam, Netherlands.


Abstract Submission:

Device aims to help kids born with congenital heart defects, like Jimmy Kimmel’s son

Late night funnyman Jimmy Kimmel made headlines last year when he choked up on air discussing his newborn’s heart condition, thrusting his son’s congenital heart defect into the national spotlight.

Meanwhile, doctors on the other side of the country are testing the latest medical advancements to help all children born with the same heart condition, using treatment that’s as non-invasive as possible.

Kimmel revealed on “Jimmy Kimmel Live!” that his now 1-year-old son, Billy, was born with a heart defect and had to undergo emergency lifesaving surgery at just three days old. Seven months later, Billy had to undergo a second heart surgery.

Kimmel brought Billy to work during an emotional episode of his typically lighthearted show after Billy’s second heart surgery.

“Daddy cries on TV, but Billy doesn’t,” he said, carrying his son on stage.

Jimmy Kimmel posted this image to his Instagram account on Oct. 19, 2017.

Jimmy Kimmel/Instagram

Jimmy Kimmel posted this image to his Instagram account on Oct. 19, 2017.

The comedian’s candid discussion of his son’s health battle thrust Billy’s condition, known as tetralogy of Fallot, into the national spotlight. Kimmel’s passionate monologue also called on all politicians to make sure that Americans, especially those with pre-existing conditions, have access to medical care.

Mac Grieb, 17, of New Jersey was born with the same heart defect as Billy Kimmel.

Mac told ABC News that he had two surgeries when he was little, and when his heart started giving him trouble again two years ago, the prospect of undergoing another open heart surgery was petrifying.

“I’ve faced life-and-death situations,” he said. “If I had to do that again, I don’t know if I could.”

Mac Grieb, now 17, was born with the same heart defect as Jimmy Kimmel's son, Billy.

Stacy Grieb

Mac Grieb, now 17, was born with the same heart defect as Jimmy Kimmel’s son, Billy.

Dr. Matthew Gillespie of the Children’s Hospital of Philadelphia told ABC News that open heart surgery is a “long” and “very invasive” procedure.

“It usually requires a week or more of recovery in the hospital,” he added.

Gillespie has been spearheading new medical research that helps children with heart defects like Billy Kimmel’s avoid surgery by using a new “Harmony Valve,” which is now in trials at a few U.S. hospitals.

The so-called Harmony Valve is inserted through a catheter and slides into position on the heart, all through a small incision in the groin.

We’re really in the midst of what I think is one of the most important breakthroughs in congenital heart disease management.

“We’re really in the midst of what I think is one of the most important breakthroughs in congenital heart disease management,” Gillespie said.

“You are putting it in without having to go in and open up the heart and sew it in,” he added. “It’s all done through catheters, and it’s engineered to stay in that position.”

Mac told ABC News that his Harmony Valve procedure was a success, and his hospital stay afterwards was the shortest it’s ever been: He left the next day.

“I went home, and I just chilled out like it was a regular day,” the teen said.

ABC News chief medical correspondent Dr. Jennifer Ashton said doctors don’t always know why a baby is born with a congenital heart defect, but that they tend to run in families.

There are also many different types of congenital heart defects and the level of severity varies greatly.

Many of these can also be picked up on prenatal ultrasounds, though some congenital heart defects are not picked up until hours or weeks after birth, usually when the baby has a cyanotic — turning blue due to Jimmy Kimmel is pictured with his son Billy on "Jimmy Kimmel Live!", Dec. 11, 2017.poor oxygenation — episode, according to Ashton.

If your child has a congenital heart defect, Ashton stressed that the best treatment option for your child really depends on the severity or the type.

Some babies require major open-heart surgery immediately after birth, some need staged operations and still others can have a minimally invasive procedure that treats the affected valve through a tiny incision.

Interventional Cardiology

Interventional cardiology is a branch of cardiology that deals specifically with the catheter-based treatment of structural heart diseases. Andreas Gruentzig is considered the father of interventional cardiology after the development of angioplasty by interventional radiologist Charles Dotter.

A large number of procedures can be performed on the heart by catheterization. This most commonly involves the insertion of a sheath into the femoral artery (but, in practice, any large peripheral artery or vein) and cannulating the heart under X-ray visualization (most commonly fluoroscopy). The radial artery may also be used for cannulation; this approach offers several advantages, including the accessibility of the artery in most patients, the easy control of bleeding even in anticoagulated patients, the enhancement of comfort because patients are capable of sitting up and walking immediately following the procedure, and the near absence of clinically significant sequelae in patients with a normal Allen test. Downsides to this approach include spasm of the artery and pain, inability to use larger catheters needed in some procedures, and more radiation exposure.


Submit your abstracts and register for the Global Congress on Cardiology and Interventional Cardiology.

Sports Cardiology

The Emerging World of Sports Cardiology

Globally we hold our athletes in the highest regard. Every four years, we send our greatest athletes to represent our country at the Olympic Games. For many of us, the athlete is the epitome of physical health and fitness. However, just like any of us, athletes can suffer from heart disease.

For the young athlete, typically those individuals under age 35, these diseases are most commonly congenital conditions that cause abnormalities in the thickness of the heart muscle, the origins of the heart arteries, or changes in the electrical system of the heart that predispose them to arrhythmias. In the more mature individual, heart disease comes in the form of common disorders such as coronary artery disease, high blood pressure, and arrhythmias such as atrial fibrillation

It takessuperpowers...

I am always surprised that many accomplished endurance athletes have stories filled with lifestyle choices that include poor diets, toxic habits like smoking, and significant family history of heart disease. In fact, studies of marathon runners find that a disproportionate number are former smokers. In fact, these underlying predispositions are often the reason that many athletes turn to endurance training in their 40s and 50s. Unfortunately, many of the lifestyle choices we make in our 20s and 30s affect our heart risk throughout life.

Cardiology 2018 provides a platform to share your view along with the experts and also provide solutions to some of the problems faced in the field of Cardiology and Interventional Cardiology.



Targeting ‘microtubules’ could prevent heart failure-Rachel Sokol

Researchers publish a study in the journal Nature Medicine that determined the cause of “stiff heart.” The findings could help to prevent future cases of heart failure.

Microtubule illustration

Microtubules (depicted here) may be key to the future treatment of heart failure.

One of the most common causes of congestive heart failure is “stiff heart syndrome.”

According to Dr. Jerry Sokol — a cardiologist in Deer Park, NY — this causes fluid to build up and back up into the lungs.

This occurs “usually in patients older than age 60,” he says.

At the microcellular level, they revealed that stiff heart appears to be related to microtubules in the cells of the heart muscle.

By treating these microtubules with newly developed research and medications, cardiac surgeons will soon be able to more effectively treat patients with this type of congestive heart failure.

The new study was led by Dr. Ben Prosser — an assistant professor of physiology in the Perelman School of Medicine at the University of Pennsylvania in Philadelphia — and is a “continuation” of research carried out 2 years ago into how microtubules help to regulate heartbeat.

Normally, microtubule cells in the heart have diverse structural and signaling roles. When these microtubules are altered, the result is believed to trigger heart disease.

Detyrosinated microtubules provide resistance that could “impede the motion of contracting heart muscle cells.” Dr. Prosser and his team tested single heart muscle cells to identify changes to the cell’s microtubules network and their consequences for normal heart function. Recent studies have suggested that chemical changes to the microtubules called detyrosination — that is, the removal of a tyrosine chemical group — control a person’s heartbeat.

The analyzed tissues from the left ventricle of heart transplant patients revealed a consistent level of proteins that resulted in the stiffening of microtubules.

Thanks to super-resolution imaging, the researchers also found a “dense, heavily detyrosinated microtubule network in the diseased heart muscle cells.”

According to Dr. Sokol, unlike the usual type of congestive heart failure — typically caused by a weakened heart muscle (when the heart doesn’t contract well after pumping) — a stiff heart resulting in heart failure occurs because the heart doesn’t “relax” well after contracting.

“Also,” he says, “the more damaged [microtubules] one has, the weaker the heart. When the damaged microtubules are compressed, the heart functions better.”

Prior clinical data from the institution identified a “direct correlation between excess microtubule detyrosination and a decline in heart function” in patients who are living with hypertrophic cardiomyopathy.

In this condition, the thickened heart muscle can lead to problems in maintaining both proper blood pressure levels and blood flow through one’s heart.

Information from transplants

For this study, the team cited research conducted by transplant cardiologist and study co-author Dr. Ken Margulies, a professor of cardiovascular medicine.

Dr. Margulies compared human heart tissue donated by heart transplant patients with normal heart tissue from other donors. The result was that detyrosination was greater in diseased hearts.

Therefore, cells from diseased hearts contain more microtubules, and these microtubules possess greater detyrosination.

This process meant impaired function in this patient population; their hearts, prior to transplant, had a lower ejection fraction (a marker of heart health designed to measure the blood pumped out of heart ventricles with every contraction) that matched up with greater detyrosination.

Currently, the team is seeking ways to target only heart muscle cell microtubules. By utilizing the Penn Gene Vector Core, the scientists are refining gene therapy approaches to transport “an enzyme to the heart that reverses detyrosination within heart muscle cells.”

Dr. Sokol adds, “Congestive heart failure is one of the most serious types of heart disease and increasingly common with age in both men and women.”

“This new research from Penn is in infancy stages,” he concludes, “but will hopefully prevent congestive heart failure in patients, resulting in healthier lives.”